Recombinant Human Sulfite oxidase, mitochondrial (SUOX), Homo sapiens (Human), E.coli, N-terminal 6xHis-SUMO-tagged, 67.6kDa, purity >90% as determined by SDS-PAGE.
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Recombinant Human Sulfite oxidase, mitochondrial (SUOX)

Recombinant Human Sulfite oxidase, mitochondrial (SUOX) — Catalyzes the oxidation of sulfite to sulfate, the terminal reaction in the oxidative degradation of sulfur-containing amino acids. Purity >90%.

SKU: BCRECP-001718 Categories: ,
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Product Specifications

Product SkuBCRECP-001718
Product DescriptionRecombinant Human Sulfite oxidase, mitochondrial (SUOX) Protein is expressed from E.coli with N-terminal 6xHis-SUMO-tagged. It contains 80-545aa. [Accession | P51687].
Uniprot No.P51687
Gene NamesSUOX
PurityGreater than 90% as determined by SDS-PAGE.
Expression SystemE.coli
Expression Region80-545aa
SpeciesHomo sapiens (Human)
Tag InfoN-terminal 6xHis-SUMO-tagged
Molecular weight67.6kDa
ActivityPlease contact us to obtain bioactivity data.
BufferIf the delivery form is liquid, the default storage buffer is Tris/PBS-based buffer, 5%-50% glycerol. If the delivery form is lyophilized powder, the buffer before lyophilization is Tris/PBS-based buffer, 6% Trehalose.
StorageStore at -20°C/-80°C upon receipt, aliquoting is necessary for mutiple use. Avoid repeated freeze-thaw cycles.
Research AreasMetabolism

Function

Catalyzes the oxidation of sulfite to sulfate, the terminal reaction in the oxidative degradation of sulfur-containing amino acids. Can run in reverse direction and reduce nitrite to nitric oxide under physiologically relevant conditions, such as hypoxia.

Biological Context

Subcellular Location: Mitochondrion intermembrane space
Disease Association: Sulfite oxidase deficiency, isolated (ISOD) : A life-threatening, autosomal recessive neurometabolic disorder characterized by severe neurological impairment. Classic ISOD manifests in the first few hours to days of life and is characterized by intractable seizures, feeding difficulties, rapidly progressive encephalopathy, microcephaly, and profound intellectual disability. Children usually die during the first few months of life. Mild ISOD manifests in infancy or early childhood and is characterized by ectopia lentis that is variably present, developmental delay and regression, movement disorder characterized by dystonia and choreoathetosis, ataxia, and rarely acute hemiplegia due to metabolic stroke. [The disease is caused by variants affecting the gene represented in this entry]
Pathway: Energy metabolism; sulfur metabolism

Product Specifications

Recombinant Human Sulfite oxidase, mitochondrial (SUOX) is a recombinant protein from Homo sapiens (Human), expressed in E.coli, covering amino acids 80-545aa, with N-terminal 6xHis-SUMO-tagged tag, molecular weight 67.6kDa, purity Greater than 90% as determined by SDS-PAGE.. Suitable for ELISA and Western Blot applications.

Recommended applications depend on the target, product format, and product-specific validation data. Refer to the product datasheet or Certificate of Analysis (CoA), or contact BioCrest Sci for application guidance.

Biological Activity: Please contact us to obtain bioactivity data.

Storage: Store at -20°C/-80°C upon receipt, aliquoting is necessary for mutiple use. Avoid repeated freeze-thaw cycles.

Shipping & Lead Time: Availability, shipping conditions, and estimated lead time are confirmed in the formal quotation or order confirmation based on the product format, destination, and order requirements.

Related products

Protein Biology

Function

Catalyzes the oxidation of sulfite to sulfate, the terminal reaction in the oxidative degradation of sulfur-containing amino acids (By similarity). Can run in reverse direction and reduce nitrite to nitric oxide under physiologically relevant conditions, such as hypoxia (PubMed:41337830)

Subcellular Location

Mitochondrion intermembrane space

Disease Association

Sulfite oxidase deficiency, isolated (ISOD) : A life-threatening, autosomal recessive neurometabolic disorder characterized by severe neurological impairment. Classic ISOD manifests in the first few hours to days of life and is characterized by intractable seizures, feeding difficulties, rapidly progressive encephalopathy, microcephaly, and profound intellectual disability. Children usually die during the first few months of life. Mild ISOD manifests in infancy or early childhood and is characterized by ectopia lentis that is variably present, developmental delay and regression, movement disorder characterized by dystonia and choreoathetosis, ataxia, and rarely acute hemiplegia due to metabolic stroke. [The disease is caused by variants affecting the gene represented in this entry]

Subunit

Homodimer

Pathway

Energy metabolism; sulfur metabolism

Gene: SUOX  |  Organism: Homo sapiens  |  Synonyms: Nitrite reductase, mitochondrial
Key Publications

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Availability, dispatch timing, shipping conditions, and delivery estimates depend on the product format, destination, and order requirements. These details are confirmed in the formal quotation or order confirmation.

How should this product be stored?

Follow the storage and handling instructions in the product-specific datasheet and Certificate of Analysis (CoA). Requirements may vary by product form and formulation; contact BioCrest Sci if product-specific guidance is needed.

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Quality-control data such as identity, purity, endotoxin level, or biological activity are provided only when specified on this product page, the datasheet, or Certificate of Analysis (CoA). Biological activity is not implied unless explicitly stated.

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